Metabolic Alterations related to T2D are very well characterised by epidemiological and research based experiments. The pathogenesis and progression of T2D is ascribed to four mechanisms; increased State-of-the-art glycation finish products (AGE) development, enhanced polyol pathway flux, activation of protein kinase C (PKC) isoforms, and improved hexosamine pathway flux[seven]. Till https://www.directivepublications.org/journal-of-diabetology-research/